FACULTY
 

Dennis M. Feeney

Professor Emeritus
Office: Logan Hall 150
Mobile Phone: 505-250-4475

Degree Received
Ph.D., University of California - Los Angeles, 1968

Research Interests
Rehabilitation pharmacology, refining a treatment to promote recovery of function after brain damage; mechanisms of traumatic brain injury and stroke, posttraumatic epilepsy.

List of Recent Publications
  • Phillips, J., Devier, D., & Feeney, D. M. (2003). Rehabilitation Pharmacology and Treatment of Brain Injury: Bridgin Laboratory Studies to Clinical Practice. Journal of Head Trauma Rehabilitation, 18(4), 342-356.
  • Sutton, R. L., Chen, M. J., Hovda, D. A., & Feeney, D. M. (2000). Alleviation of brain injury-induced cerebral metabolic depression by amphetamine: A cytochrome oxidase histochemistry study. Neural Plasticity, 7, 109-126.
  • Gasparovic, C., King, D., & Feeney, D. M. (1999). Metabolism in single rat brain slices measured by magnetic resonance spectroscopy. Brain Research Protocols, 4, 97-102.
  • Feeney, D. M. (1998). Guest Editorial: Enhanced neocortical neural sprouting, synaptogenesis, and behavioral recovery with d-amphetamine therapy after neocortical infarction in rats. Stroke, 29, 2381-2395.
  • Feeney, D. M. (1998). Mechanisms of noradrenergic modulation of physical therapy: Effects on functional recovery after cortical injury. In L. Goldstein (Ed.), Restorative Neurology: Advances in the Pharmacology of Recovery After Stroke. Armonc, NY: Futura Publishing Co., 35-78.
  • Feeney, D. M. (1998). Rehabilitation Pharmacology: Noradrenergic Enhancement of Physical Therapy. In M. Ginsberg & J. Bogousslavsky (Eds.), Cerebrovascular Disease: Pathophysiology, Diagnosis and Management. Vol. I., Chapter 44. Cambridge MA: Blackwell Scientific Press, 620-636.
  • Feeney, D. M., Gullotta, F. P., & Gilmore, W. (1998). Hyposexuality accompanies temporal lobe epilepsy in the cat. Epilepsia, 39, 140-144.
  • Feeney, D. M. (1997). From laboratory to clinic: Noradrenergic enhancement of physical therapy for stroke or trauma patients. In Freund, H. J., Sabel, B. A., & Witte, O. W. (Eds.), Brain Plasticity: Advances in Neurology, 73, Philadelphia: Lippencott-Raven Publishers, 383-394.
  • Queen, S., Chen, M., & Feeney, D. M. (1997). Amphetamine alleviates the reduced local cerebral glucose utilization produced by traumatic brain injury. Brain Research, 777, 42-50.
  • Feeney, D. M. (1996). Brain damage in a traumatic brain injury model: Noradrenergic pharmacotherapy promotes functional recovery. In J. F. Toole & D. C. Good (Eds.), Neuroimaging in Rehabilitation: Comprehensive Neurological Rehabilitation, Vol. 9. New York: Demos Vermande, 91-124.
  • Queen, S., & Feeney, D. M. (1996). Cerebral trauma produces transient hypermetabolism. Brain Research, 724, 246-250.
  • Feeney, D. M., Weisend, M. P., & Kline, A. E. (1994). Noradrenergic pharmacotherapy, intracerebral infusion and adrenal transplantation promote functional recovery after cortical damage. Journal of Neurotransplantation and Plasticity, 4, 199-214.
  • Kline, A. E., Chen, M. J., Tso-Oliveras, D. Y., & Feeney, D. M. (1994). Methylphenidate treatment following ablation induced hemiplegia: Experience during drug action alters effects on recovery of function. Pharmacology, Biochemistry and Behavior, 48, 773-779.
  • Krobert, K. A., Sutton, R. L., & Feeney, D. M. (1994). Spontaneous and amphetamine evoked release of noradrenaline after cortical contusion. Journal of Neurochemistry, 62, 2233-2240.
  • Sutton, R. L., & Feeney, D. M. (1994). Noradrenergic pharmacotherapy and functional recovery after cortical injury. In L. S. Illis (Ed.), Neurological Rehabilitation. Oxford, England: Blackwell Publishing Co., 469-480.
  • Weisend, M. P. & Feeney, D. M. (1994). The relationship between traumatic brain injury-induced changes in brain temperature and behavioral and anatomical outcome. Journal of Neurosurgery, 80, 120-132.
  • Sutton, R. L., & Feeney, D. M. (1992). Alpha-noradrenergic agonists and antagonists affect recovery and maintenance of beam-walking ability after sensorimotor cortex ablation in the rat. Restorative Neurology and Neuroscience, 4, 1-11.
  • Boyeson, M. G., & Feeney, D. M. (1991). Adverse effects of catecholaminergic drugs following unilateral cerebellar ablations. Restorative Neurology and Neuroscience, 3, 227-233.
  • Boyeson, M. G., Feeney, D. M., & Dail, W. G. (1991). Cortical microstimulation thresholds adjacent to sensorimotor cortex injury. Journal of Neurotrauma, 8, 205-217.
  • Feeney, D. M.. (1991). Pharmacologic modulation of recovery after brain injury: A reconsideration of diaschisis. Journal of Neurologic Rehabilitation, 5, 113-128.
  • Feeney, D. M., Weisend, M. P., Krobert, K., & Queen, S. (1991). Feasibility study of pharmacological treatment to reduce morbidity and mortality after brain injury. Technical Report, Contract No. DAMD17-86-C-6144, University of New Mexico, April 2, 1991. NTIS, ADA 239283.
  • Boyeson, M. G., & Feeney, D. M. (1990). Intraventricular norepinephrine facilitates recovery following sensorimotor cortex injury. Pharmacology, Biochemistry and Behavior, 35, 497-501.
  • Feeney, D. M., & Westerberg, V. S. (1990). Norepinephrine and brain damage: Alpha noradrenergic pharmacology alters functional recovery after cortical trauma. Canadian Journal of Psychology, 44, 233-252.

Other information
Professor of Psychology & Physiology
Research collaboration with the following departments in our medical school: Anatomy, Neurology, and Neurosurgery.